· autoimmune disease
· aetiology unknown
· thought to be combination of
· external trigger (maybe infective agent)
· genetic susceptibility
· body produces antibodies against is own immunoglobulins
· antibodies called antiglobulins
· directed against Fc region of IgG
General
· exogenous trigger produces antigen
· antigen somehow alters IgG to become antigenic
· helper T cells activate B cells to become plasma cells
· plasma cells produce immunoglobulins (RFs) directed against IgG
· T cells and B cells chronically antigenically challenged
· unknown why RF continues to be produced
Specific
· in RA, synovium acts as lymphoid organ
· local helper T cells activated
· local plasma cells produce RF
· antibody-antiglobulin complexes formed
· immune complexes stimulate complement
· inflammatory response occurs
· are antiglobulins produced in RA
· RFs are heterogenous
· may be IgM, IgG, gA or IgE
· present in 80% of patients with RA
· may also be present in
· infectious and autoimmune diseases
· hyperglobulinaemias
· B-cell lymphoproliferative disorders
· elderly patients
· RF secreted by plasma cells in synovium
· high titre RFs associated with more severe disease
· role of RF in pathogenesis unclear
Measurement
· classically use agglutination methods
· most sensitive for IgM
· usually measured with latex aggregation test
· latex particles coated with human IgG
· particles visibly aggregate in presence of RF
· titre is highest dilution of serum that causes agglutination
· titre > 1:80 considered positive
· no obvious abnormalities in RA
· may be deficiency in T suppressor cells
· infectious agent could operate via one of four mechanisms
1. agent or derived antigens within joint space causes immune response
· eg. rubella
2. agent or derived antigens at distant site but immune response causes arthritis
· eg. Reiter’s syndrome
3. multiplication of agent in joint space
· eg. pyogenic infection
4. agent produces arthitogenic toxins
· no example known
· no definite organism or vector found yet
· associations found with histocompatibility antigens
· patients with RA have increased incidence of HLA DRW4
· may reflect genetic susceptibility to develop autoimmune disease